Mitochondria-Supported Skin Rejuvenation & Redox Control Stack
Mechanistic Synergy for Research:
The inclusion of NAD⁺ (Nicotinamide Adenine Dinucleotide) adds metabolic restoration and DNA repair functionality to the antioxidant and regenerative foundation of GHK-Cu and Glutathione.
- Cellular Energy & Mitochondrial Optimization:
NAD⁺ boosts oxidative phosphorylation, supplying ATP required for collagen production, DNA repair, and skin turnover in dermal fibroblasts. This is crucial in aging skin models with reduced metabolic output. - DNA Repair & Longevity Pathway Activation:
NAD⁺ fuels PARP and sirtuin enzyme activity, both of which mediate DNA repair and delay cellular senescence. When combined with GHK-Cu’s regenerative cues, the result is improved tissue functionality and reduced senescent cell accumulation. - Extended Antioxidant Capacity:
NAD⁺ supports glutathione recycling via NADPH regeneration, prolonging GSH’s protective function during oxidative insult and inflammation. This maintains redox homeostasis in skin, scalp, and vascular endothelium. - Synergistic Support of Dermal Cell Renewal:
The trio stimulates fibroblast proliferation, ECM deposition, mitochondrial repair, and oxidative protection, enabling robust study of skin healing and anti-aging models.
Best suited for:
Advanced dermal research involving oxidative damage, age-related collagen loss, mitochondrial dysfunction, or UV/photoaging models.
